在期刊编辑出版gydF4y2Ba

1。而Kalafatis, m .识别des fonctionnels du因素Willebrand humain l 'aide d 'anticorps monoclonaux。gydF4y2Ba这些德德因为学校大Biochimie巴黎VIgydF4y2Ba(gydF4y2Ba博士学位。gydF4y2Ba1989年巴黎大学生物化学)。gydF4y2Ba

2。吉尔马,摩根大通,Kalafatis, M。Pietu, G., Lavergne, J.M., Chopek, M.W., Edgington, T.S., and Meyer, D. Mapping of distinct von Willebrand Factor domains interacting with platelet GPIb and GPIIb/IIIa and with collagen using monoclonal antibodies.血gydF4y2Ba,1986,67,1356 - 1366gydF4y2Ba

3所示。Kalafatis, M。高桥,Y。吉尔马,摩根大通,和Meyer, D. Localization of a collagen interactive domain of human von Willebrand Factor between amino acid residues Gly 911 and Glu 1365.血gydF4y2Ba,1987,70,1577 - 1583。gydF4y2Ba

4所示。高桥,Y。,Kalafatis, M。吉尔马,摩根大通,Sewerin, K., Andersson, L.O., and Meyer, D. Localization of a Factor VIII binding domain on a 34 kDa fragment of the N-terminal portion of von Willebrand Factor.血gydF4y2Ba,1987,70,1679 - 1682。gydF4y2Ba

5。高桥,Y。,Kalafatis, M。吉尔马,摩根大通,和Meyer, D. Abnormality of the N-terminal portion of von Willebrand Factor in type IIA and IIC von Willebrand disease.Thromb。HaemostgydF4y2Ba。gydF4y2Ba,1988,498 - 505。gydF4y2Ba

6。Kalafatis, M。Jenny, R.J., Mann, and K.G. Identification and characterization of a phospholipid-binding site of bovine factor Va.生物。化学gydF4y2Ba。gydF4y2Ba,1990,265,21580 - 21589。gydF4y2Ba

7所示。Kalafatis, M。兰德,医学博士,Jenny, R.J., Ehrlich, Y.H., and Mann, K.G. Phosphorylation of factor Va and VIIIa by activated platelets.血gydF4y2Ba,1993,81,704 - 719。gydF4y2Ba

8。天雪,J。,Kalafatis, M。和曼,kg。Determination of the disulfide bridges in factor Va light chain.生物化学gydF4y2Ba,1993年,32岁,5917 - 5923。gydF4y2Ba

9。Kalafatis, M。和曼,kg。Role of the membrane in the inactivation of factor Va by activated protein C.生物。化学gydF4y2Ba,1993,268,27246 - 27257。gydF4y2Ba

10。Kalafatis, M。兰德,医学博士,和曼,kg。Factor Va-membrane interaction is mediated by two regions located on the light chain of the cofactor.生物化学gydF4y2Ba,1994,33岁,486 - 493。gydF4y2Ba

11。Butenas, S。,Lawson, J.H, Kalafatis M., and Mann, K.G. Cooperative interaction of divalent metal ions, substrate, and tissue factor with factor VIIa.生物化学gydF4y2Ba1994 33,3449 - 3456。gydF4y2Ba

12。兰德,医学博士,Kalafatis, M。和曼,kg。Platelet coagulation factor Va: The major secretory platelet phosphoprotein.血gydF4y2Ba,1994,83,2180 - 2190。gydF4y2Ba

13。Kalafatis, M。天雪,J。,Lawler, C.M., and Mann, K.G. Contribution of the heavy and light chains of factor Va to the interaction with factor Xa.生物化学gydF4y2Ba,1994,33岁,6538 - 6545。gydF4y2Ba

14。陆,D。,Kalafatis, M。曼,K。G., and Long, G. L. Loss of membrane-dependent factor Va cleavage: A mechanistic interpretation of the pathology of protein C_{佛蒙特州gydF4y2Ba}。gydF4y2Ba血gydF4y2Ba,gydF4y2Ba1994,84,687 - 690。gydF4y2Ba

15。劳森,J.H.,Kalafatis, M。Stram, S., and Mann, K.G. A model for the tissue factor pathway to thrombin. 1. An empirical study.生物。化学gydF4y2Ba。gydF4y2Ba,1994,269,23357 - 23366。gydF4y2Ba

16。天雪,J。,Kalafatis, M。问题,jr宫,C。,和曼,kg。Determination of the disulfide bridges location in bovine factor Va heavy chain.生物化学gydF4y2Ba1994 33,13109 - 13116。gydF4y2Ba

17所示。Kalafatis, M。兰德,医学博士,和曼,kg。的mechanism of inactivation of human factor V and human factor Va by activated protein C.生物。化学gydF4y2Ba。gydF4y2Ba,1994,269,31869 - 31880。gydF4y2Ba

18岁。Kalafatis, M。Bertina, R.M., Rand, M.D., and Mann, K.G. Characterization of the molecular defect in factor V^{R506QgydF4y2Ba}。gydF4y2Ba生物。化学gydF4y2Ba。gydF4y2Ba,1995,270,4053 - 4057。gydF4y2Ba

19所示。Kalafatis, M。和曼,kg。Factor V Leiden and Thrombophilia.心血管病。j .地中海。gydF4y2Ba,1995,332,1382 - 1383。gydF4y2Ba

20.Camire,智慧化,Kalafatis, M。Cushman, M., Tracy, R.P., Mann, K.G., and Tracy, P.B. The mechanism of inactivation of platelet-derived factor Va from normal and APC-resistant individuals by activated protein C.生物。化学gydF4y2Ba,1995,270,20794 - 20800。gydF4y2Ba

21。Kalafatis, M。陆,D。,Bertina, R.M., Long, G.L., and Mann, K.G. Biochemical prototype for familial thrombosis: A study combining a functional protein C mutation and factor V Leiden.Arterioscler。Thromb。Vasc。医学杂志。gydF4y2Ba1995年,15日,2181 - 2187。gydF4y2Ba

22。Kalafatis, M。Haley, P.E., Lu, D., Bertina, R.M., Long, G.L., and Mann, K.G. Proteolytic events that regulate factor V activity in whole plasma from normal and activated protein C (APC)-resistant individuals during clotting: An insight into the APC-resistance assay.血gydF4y2Ba,1996,87,4695 - 4707。gydF4y2Ba

23。陆,D。,Kalafatis, M。曼,kg。,和长,基准线Comparison of activated protein C/protein S-mediated inactivation of human factor VIII and factor V.血gydF4y2Ba,1996,87,4708 - 4717。gydF4y2Ba

24。Simioni, P。,Kalafatis, M。Millar D.S., Henderson, S.C., Luni, S., Cooper, D.N., and Girolami, A. Compound heterozygous protein C deficiency resulting in the presence of only thebgydF4y2Ba的形式在血浆蛋白C。gydF4y2Ba血gydF4y2Ba,1996,88,2101 - 2108。gydF4y2Ba

25。Bajzar, L。,Kalafatis, M。Simioni, P。,和Tracy, P.B. An antifibrinolytic mechanism describing the prothrombotic effect of factor V^{莱顿gydF4y2Ba}。gydF4y2Ba生物。化学gydF4y2Ba,1996,271,22949 - 22952。gydF4y2Ba

26岁。Butenas, S。,Kalafatis M., and Mann, K.G. Analysis of tissue plasminogen activator specificity using peptidyl fluorogenic substrates.生物化学gydF4y2Ba,gydF4y2Ba1997年,36岁,2123 - 2131。gydF4y2Ba

27。范' t转向,C。金,新泽西州,Kalafatis, M。和曼,kg。在hibitory mechanism of the protein C pathway on tissue factor induced thrombin generation. Synergistic effect in combination with tissue factor pathway inhibitor.生物。化学gydF4y2Ba,1997,272,7983 - 7994。gydF4y2Ba

28。曼,kg。,Hockin,年报,Begin, K.J., and Kalafatis, M. Activated protein C cleavage of factor Va leads to dissociation of the A2 domain.生物。化学gydF4y2Ba,1997,272,20678 - 20683。gydF4y2Ba

29。范' t转向,C。Kalafatis, M。伯蒂娜,智慧化Simioni, P。,和曼,kg。在creased tissue factor-initiated prothrombin activation as a result of the Arg^{506年gydF4y2Ba}®gydF4y2BaGln突变因子VgydF4y2Ba^{莱顿gydF4y2Ba}。gydF4y2Ba生物。化学gydF4y2Ba,1997,272,20721 - 20729。gydF4y2Ba

30.伊根,J.O.,Kalafatis, M。和曼,kg。的effect of Arg^{306年gydF4y2Ba}®gydF4y2Ba阿拉巴马州和参数gydF4y2Ba^{506年gydF4y2Ba}®gydF4y2Ba弗吉尼亚州Gln替换重组体人失活的因素通过活化蛋白C和蛋白S。gydF4y2Ba蛋白质科学gydF4y2Ba,gydF4y2Ba1997年,2016 - 2027。gydF4y2Ba

31日。范' t转向,C。金,新泽西州,Kalafatis, M。Simioni, P。,Bertina R.M., and Mann, K.G. An in vitro analysis of the combination of factor V^{莱顿gydF4y2Ba}血友病。gydF4y2Ba血gydF4y2Ba,gydF4y2Ba1997,90,3067 - 3072。gydF4y2Ba

32。Hockin,年报,Kalafatis, M。Shatos, M.A., and Mann, K.G. Protein C activation and factor Va inactivation on human umbilical vein endothelial cells.Arterioscler。Thromb。Vasc。医学杂志。gydF4y2Ba,gydF4y2Ba1997年,17岁,2765 - 2775。gydF4y2Ba

33。Kalafatis, m .识别和局部特征因子Va重chain-kinase从人类血小板。gydF4y2Ba生物。化学gydF4y2Ba,1998,273,8459 - 8466。gydF4y2Ba

34。Camire,智慧化,Kalafatis, M。Simioni, P。,Girolami, A., and Tracy, P.B. Platelet-derived factor Va/Va^{莱顿gydF4y2Ba}代数余子式活动持续激活血小板,尽管表面活化蛋白C的存在。gydF4y2Ba血gydF4y2Ba1998,91,2818 - 2829。gydF4y2Ba

35。长,基准线陆,D。谢,R。,和Kalafatis, M。Human protein S cleavage and inactivation by coagulation factor Xa.生物。化学gydF4y2Ba,1998,273,11521 - 11526。gydF4y2Ba

36。Camire,智慧化,Kalafatis, M。和Tracy, P.B. Proteolysis of factor V by cathepsin G and elastase indicates that cleavage at Arg^{1545年gydF4y2Ba}优化代数余子式Xa绑定函数通过促进因素。gydF4y2Ba生物化学gydF4y2Ba,gydF4y2Ba1998年,37岁,11896 - 11906。gydF4y2Ba

37岁。Castoldi E。,Kalafatis, M。Lunghi, B., Simioni, P., Ioannou, P.A., Petio, M., Girolami, A., Mann, K.G., and Bernardi, F. Molecular bases of pseudo-homozygous APC resistance: The compound heterozygosity for FV R506Q and a FV null mutation results in the exclusive presence of FV Leiden molecules in plasma.Thromb。Haemost。gydF4y2Ba1998,80,403 - 406。gydF4y2Ba

38。Simioni, P。,Kalafatis, M。Manfrin, D., Tormene, D., and Girolami, A. Factor V variants, activated protein C resistance and venous thromboembolism.血液Coagul.Fibrinolysis。gydF4y2Ba1998、9、661 - 2。gydF4y2Ba

39岁。Kalafatis, M。Bernardi, F., Simioni, P., Lunghi B., Girolami, A and Mann, K.G. Phenotype and genotype expression in pseudohomozygous factor V^{莱顿gydF4y2Ba}。表型分析的必要性。gydF4y2BaArterioscler。Thromb。Vasc。医学杂志。gydF4y2Ba1999年,19岁,336 - 342。gydF4y2Ba

40。Hockin,年报,Cawthern, K.M., Kalafatis, M., and Mann, K.G. A model describing the inactivation of factor Va by activated protein C (APC): Bond cleavage, fragment dissociation and product inhibition.生物化学gydF4y2Ba1999年,38岁,6918 - 6934。gydF4y2Ba

41岁。Castoldi E。,Simioni, P。,Kalafatis, M。Lunghi, B., Tormene, D., Girelli, D., Girolami, A., and Bernardi, F. Combinations of four mutations (FV R506Q, FVH1299R, FVY1702C, PT20210G/A) affecting the prothrombinase complex in a thrombophilic family.血gydF4y2Ba2000,96,1443 - 1448。gydF4y2Ba

42。Simioni, P。,Vianello, F., Kalafatis, M., Barzon, L., Ladogana, S., Paolucci, P., Carotenuto, M., Dal Bello, F., Palù, P., and Girolami A. A dysfunctional factor X (Factor X San Giovanni Rotondo) Present at Homozygous and Double heterozygous level: Identification of a novel micro-deletion (delC556) and missense mutation (Lys^{408年gydF4y2Ba}®gydF4y2BaAsn) X基因因素gydF4y2Ba。gydF4y2Ba一个意大利家庭的研究。gydF4y2BaThromb.Res。gydF4y2Ba2001 101,219 - 230。gydF4y2Ba

43。Kalafatis、m和Mann kg。膜的作用Va由血纤维蛋白溶酶失活的因素。gydF4y2Ba氨基酸地区307 - 348 V的因素起着关键作用的因素Va代数余子式函数gydF4y2Ba。gydF4y2Ba生物。化学。gydF4y2Ba2001,276,18614 - 18623。gydF4y2Ba

44岁。Kalafatis, M。Simioni, P。,和Bernardi, F. Phenotype and genotype expression in pseudohomozygous R2 factor V.血gydF4y2Ba2001,98,1988 - 1989。gydF4y2Ba

45岁。Simioni, P。,Kalafatis M., Tormene D., Luni, S., Zerbinati, P., Barzon, L., Palù, G., and Girolami A. Abnormal propeptide processing resulting in the presence of two abnormal species of protein C in plasma. Characterization of the dysfunctional protein C Padua_3gydF4y2Ba(蛋白质CgydF4y2Ba_{R-1L /前肽gydF4y2Ba})。gydF4y2BaThromb。Haemost。gydF4y2Ba2001,86,1017 - 1022。gydF4y2Ba

46岁。问题,jr,Kalafatis, M。和Tracy, P.B. Carbohydrate moieties on factor V, but not the derived cofactor, factor Va, regulate its inactivation by activated protein C.生物化学gydF4y2Ba2002年,41岁,1672 - 1680。gydF4y2Ba

47岁。挂,K。,Sun, X., Ding, H., Kalafatis, M., Simioni P., and Guo B. A MALDI-TOF based mini sequencing method for screening of the G^{1691年gydF4y2Ba}®gydF4y2BaV基因突变的因素。gydF4y2Ba血液Coagul。纤维蛋白溶解。gydF4y2Ba2002年,117 - 122。gydF4y2Ba

48。Kalafatis, M。Simioni, P。,Tormene, D., Beck, D.O., Luni, S., and Girolami, A. Isolation and characterization of an anti-factor V antibody causing activated protein C resistance from a patient with severe thrombotic manifestations.血gydF4y2Ba2002,99,3985 - 3992。gydF4y2Ba

49。辛格L.S. Kalafatis, m .测序的全长cDNA编码gydF4y2Ba一个gydF4y2Ba和gydF4y2BabgydF4y2Ba子单元的人类酪蛋白激酶ⅱ从人类血小板和巨核细胞的细胞。gydF4y2Ba酪蛋白激酶ⅱ的表达gydF4y2Ba一个gydF4y2Baintronless巨核细胞的细胞系的基因。gydF4y2Ba生物化学gydF4y2Ba2002年,41岁,8935 - 8940。gydF4y2Ba

50。Kalafatis, m·贝克,交货单为凝血因子结合位点的识别Xa重链的因素。gydF4y2Ba氨基酸残基323 - 331 V的因素包含一个交互式网站激活因子XgydF4y2Ba。gydF4y2Ba生物化学gydF4y2Ba。2002年,41岁,12715 - 12728。gydF4y2Ba

51。辛格L.S.,Bukys,硕士,贝克,交货单,和Kalafatis, M。Amino acids Glu^{323年gydF4y2Ba},酪氨酸gydF4y2Ba^{324年gydF4y2Ba},GlugydF4y2Ba^{330年gydF4y2Ba}和瓦尔gydF4y2Ba^{331年gydF4y2Ba}弗吉尼亚州的因素重链代数余子式的表达活动至关重要。gydF4y2Ba生物。化学gydF4y2Ba。2003,278,28335 - 28345。gydF4y2Ba

52岁。Kalafatis, M。贝克,交货单,和曼,kg。Structural requirements for expression of factor Va activity.生物。化学gydF4y2Ba。2003,278,33550 - 33561。gydF4y2Ba

53岁。贝克,交货单,Bukys,硕士,辛格L.S.,Szabo, K.A. and Kalafatis, M. The contribution of amino acid region Asp^{695年gydF4y2Ba}酪氨酸gydF4y2Ba^{698年gydF4y2Ba}弗吉尼亚州因子V procofactor激活因子的函数。gydF4y2Ba生物。化学。gydF4y2Ba2004,279,3084 - 3095。gydF4y2Ba

54。古尔德,得到,Simioni, P。,问题,jrLuni S。,Kalafatis M., and Tracy P.B. Megakaryocytes endocytose and subsequently modify human plasma-derived factor V在活的有机体内gydF4y2Ba形成血小板源池。gydF4y2Baj . Thromb。Haemost。gydF4y2Ba2005年,450 - 456gydF4y2Ba。gydF4y2Ba

55。Bukys,硕士,Blum, M.A., Young, P.Y, Bruffato, N., Nesheim, M.E., and Kalafatis, M. Incorporation of Factor Va into Prothrombinase is required for coordinated cleavage of Prothrombin by Factor Xa.生物。化学。gydF4y2Ba2005,280,27393 - 27401。gydF4y2Ba

56。班,T。,Kalafatis, M。和Gogonea, V. Completed Three-Dimensional Model of Human Coagulation Factor Va. Molecular Dynamics Simulations and Structural Analyses.生物化学gydF4y2Ba2005年,44岁,13082 - 13090。gydF4y2Ba

57。Bukys,硕士班,T。金,P.Y.,贝克,交货单,Nesheim, M.E., and Kalafatis, M. The structural integrity of anion binding exosite I of thrombin is required and sufficient for timely activation of factor V and factor VIII.生物。化学。gydF4y2Ba2006,281,18569 - 18580。gydF4y2Ba

58岁。Butenas, S。Orfeo, T。,Kalafatis, M。和曼kg。gydF4y2BaPeptidomimetic抑制剂激活蛋白C:对血友病的管理。gydF4y2Baj . Thromb。Haemost。gydF4y2Ba2006年,2411 - 2416。gydF4y2Ba

59。Bukys,硕士金,P.Y.,Nesheim, M. E., and Kalafatis, M. A control switch for prothrombinase.Va COOH-terminus hirudin-like肽的因素重链调节速率和途径供prothrombinase凝血酶原的激活gydF4y2Ba。gydF4y2Ba生物。化学gydF4y2Ba。gydF4y2Ba2006,281,39194 - 39204。gydF4y2Ba

60。埃尔多安,E。,Bukys,硕士,Orfeo T。,曼,kg。,和Kalafatis, M。Identification of an inactivating cleavage site for thrombin on the heavy chain of factor Va.Thromb。Haemost。gydF4y2Ba2007,98,998 - 1006。gydF4y2Ba

61年。埃尔多安,E。,Bukys,硕士,和Kalafatis, M。的contribution of amino acids 1508-1515 of factor V to light chain formation.j . Thromb。Haemost。gydF4y2Ba2008年,118 - 124gydF4y2Ba

62年。Bukys硕士班,T。,金姆。P.Y.,Nesheim, M. E., and Kalafatis, M. The interaction of fragment 1of prothrombin with the membrane surface is a prerequisite for optimum expression of factor Va cofactor activity within prothrombinase.Thromb。HaemostgydF4y2Ba。gydF4y2Ba,2008,99,511 - 522。gydF4y2Ba

63年。Barhoover,硕士班,T。,贝克,交货单,Bukys,硕士,和Kalafatis, M。的contribution of amino acid region 334-335 from factor Va heavy chain to the catalytic efficiency of prothrombinase.生物化学gydF4y2Ba,2008,6840 - 6850。gydF4y2Ba

64年。Hirbawi, J。,Bukys,硕士,Barhoover,硕士埃尔多安,E。,和Kalafatis M. Role of the acidic hirudin-like COOH-terminal amino acid region of factor Va heavy chain in the enhanced function of prothrombinase.生物化学gydF4y2Ba,gydF4y2Ba2008年,47岁,7963 - 7974。gydF4y2Ba

65年。Barhoover,硕士班,T。Bukys,硕士,和Kalafatis M. Cooperative regulation of the activity of factor Xa within prothrombinase by discrete amino acid regions from factor Va heavy chain.生物化学gydF4y2Ba,2008,12835 - 12843。gydF4y2Ba

66. Hirbawi, J。,Vaughn, J.L., Bukys, M.A., Vos, H., and Kalafatis M.氨基酸的贡献因素的地区659 - 663 Va重链的活性因子在prothrombinase XagydF4y2Ba。gydF4y2Ba生物化学gydF4y2Ba,2010年,49岁,8520 - 8534。gydF4y2Ba

67年。Barhoover,硕士和Kalafatis m .劈理这两个参数gydF4y2Ba^{306年gydF4y2Ba}和参数gydF4y2Ba^{506年gydF4y2Ba}需要和满足及时、有效因子Va的失活蛋白C激活。gydF4y2BaCoag血纤维蛋白溶解gydF4y2Ba,2011,22岁,317 - 324。gydF4y2Ba

68年。Wiencek jr,Na M。,Hirbawi J., and Kalafatis, M. Amino Acid region 1000-1008 of factor V is a dynamic regulator for the emergence of procoagulant activity.生物。化学gydF4y2Ba,2013,288,37026 - 37038。gydF4y2Ba

69年。Wiencek jr,Hirbawi J。绮,风险投资,和Kalafatis, M。gydF4y2Ba氨基酸的双重监管作用Leu480 Gln481凝血酶原。gydF4y2Ba生物。化学gydF4y2Ba,2016,291,1565 - 1581。gydF4y2Ba

70年。特纳·,Lindner D.J., and Kalafatis, M. Recombinant human Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand selectively induces apoptosis in malignant melanoma.Int J癌症杂志gydF4y2Ba2017年,4 (1),1 - 8。gydF4y2Ba

71年。特纳·Kalafatis,此案的线索:此案的线索。死亡受体作为rhTRAIL-Sensitivity标记gydF4y2Ba。gydF4y2Ba应用实验医学杂志(JALM)gydF4y2Ba2017年,2 (2)176 - 185。gydF4y2Ba

72年。Hirbawi j .和Kalafatis M:gydF4y2Ba引人入胜的影响因素Va重链的酸性COOH-Terminus prothrombinase活动和功能。gydF4y2BaACSωgydF4y2Ba2017年,5529 - 5537。gydF4y2Ba

73年。Manouchehri, J.M. Kalafatis, m . rhTRAIL-Resistant三阴性乳腺癌的敏化Silibinin Co-Treatment。gydF4y2Ba抗癌的研究gydF4y2Ba2017年,gydF4y2Ba37 (12),6593 - 6599。gydF4y2Ba

74年。Manouchehri, J.M.、特纳·,和Kalafatis, M。TRAIL-induced apoptosis in TRAIL-resistant breast carcinoma through Quercetin co-treatment.乳腺癌:基础和临床研究gydF4y2Ba2018、12、1 - 12。gydF4y2Ba

75年。特纳·,Manouchehri, J.M.,和Kalafatis, M。gydF4y2Ba敏化重组人类肿瘤坏死factor-related凋亡诱导ligand-resistant恶性黑素瘤的槲皮素gydF4y2Ba黑色素瘤研究gydF4y2Ba,2018,28 (4),277 - 285。gydF4y2Ba

76年。Manouchehri, J.M. Kalafatis, m .乌索酸促进rhTRAIL-resistant的敏化三阴性乳腺癌。gydF4y2Ba抗癌的研究gydF4y2Ba2018年,gydF4y2Ba38 (12),6789 - 6795。gydF4y2Ba

77年。Kalafatis, M。gydF4y2BaCOVID-19:严重的血管疾病和呼吸系统疾病的主要症状。gydF4y2Ba应用实验医学杂志(JALM)gydF4y2Ba2021年,6 (5),1099 - 1104。gydF4y2Ba

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78年。Kalafatis, M。吉尔马,摩根大通,和Meyer, D. Identification des domaines fonctionnels du facteur Willebrand humain.创新。科技杂志。地中海。gydF4y2Ba(ITBM)gydF4y2Ba,gydF4y2Ba1988年,682 - 690。gydF4y2Ba

79年。Kalafatis, M。Swords, N.A., Rand, M.D., and Mann, K.G. Membrane-dependent reactions in blood coagulation. Role of the vitamin K-dependent enzyme complexes.疾病的分子基础gydF4y2Ba。gydF4y2BaBiochim。Biophys。学报gydF4y2Ba,1994,1227,113 - 129。gydF4y2Ba

80年。曼,K。G, Kalafatis, m .凝固爆炸。gydF4y2Ba脑血管疾病gydF4y2Ba,gydF4y2Ba1995年,93 - 97。gydF4y2Ba

81年。Kalafatis, M。伊根,J.O.,van’t Veer, C., and Mann, K.G. Regulation and regulatory role ofggydF4y2Ba-carboxyglutamic酸含有凝血因子。gydF4y2Ba真核基因表达的关键评论gydF4y2Ba1996 6 87 - 101。gydF4y2Ba

82年。Kalafatis, m . C-resistance激活蛋白的分子基础。gydF4y2Ba美国心脏协会通讯gydF4y2Ba(血栓委员会)1996年秋季,7 - 9页。gydF4y2Ba

83年。Kalafatis, M。和曼,kg。Factor V^{莱顿gydF4y2Ba}和血栓形成倾向。gydF4y2BaArterioscler。Thromb。Vasc。医学杂志。gydF4y2Ba,1997,620 - 627。gydF4y2Ba

84年。Kalafatis, M。伊根,J.O.,van’t Veer, C., Cawthern, K.M., and Mann, K.G. The Regulation of clotting factors.真核基因表达的关键评论gydF4y2Ba,1997,241 - 280。gydF4y2Ba

85年。曼,kg。Kalafatis, m .因素V: Jeckyll博士和海德先生的结合。gydF4y2Ba血gydF4y2Ba、2003、101、20 - 30。gydF4y2Ba(gydF4y2Ba图2的手稿是101卷的封面照片,1号,2003年1月1日版的血液gydF4y2Ba)gydF4y2Ba。gydF4y2Ba

86年。Kalafatis, m .因子V:大量的有效的抗凝分子。gydF4y2Ba目前在血液学的意见gydF4y2Ba,2005,141 - 148gydF4y2Ba

书的章节gydF4y2Ba

87年。吉尔马,摩根大通,Kalafatis, M。和Meyer, D. Mapping of von Willebrand Factor functional domains with monoclonal antibodies. In:血因子/血管性血友病因子。生物和临床进展gydF4y2Ba。gydF4y2BaEds。N.L. Ciavarella, Z.R.最近,T.S.齐默尔曼;1986页137 - 148。Wichtig Editore,意大利。gydF4y2Ba

88年。吉尔马,摩根大通,Kalafatis, M。高桥,Y。,和Meyer, D. Recognition of three distinct functional domains of human von Willebrand factor using monoclonal antibodies. In:利用单克隆抗体的理解和检测血小板的活动gydF4y2Ba。gydF4y2Ba埃德·j·麦格雷戈;1986年,页219 - 225。爱思唯尔科学出版商帐面价值、荷兰。gydF4y2Ba

89年。Kalafatis, M。Krishnaswamy, S., Rand, M.D., and Mann, K.G. Factor V. In:在凝固蛋白水解酶,纤维蛋白溶解,补体激活。gydF4y2Ba酶学方法gydF4y2Ba,1993,222,224 - 236。gydF4y2Ba

90年。Kalafatis, M。伊根,J.O.,和曼,kg。Coagulation Factors. In:内皮细胞在临床实践中gydF4y2BaEds。Rubanyi / Dzau 1996页245 - 264。马塞尔•德克尔公司,出版商,美国纽约。gydF4y2Ba

91年。Kalafatis, M。范' t转向,C。,和曼,kg。Coagulation Factors. In:百科全书式的参考血管生物学和病理学。gydF4y2Ba艾德。a . Bikfalvi;2000年,49 - 64页。德国斯普林格出版社柏林海德堡。gydF4y2Ba

92年。gydF4y2BaKalafatis, M。gydF4y2Ba和曼kg。gydF4y2Ba因素V: DgydF4y2Ba_rgydF4y2BaJeckyl和MgydF4y2Ba_rgydF4y2Ba海德。在gydF4y2Ba:《新世纪血友病治疗。gydF4y2BaEds。霍夫曼D.M.梦露,Hedner,核磁共振,c . Negrier G.F.萨维治,G.C.白色;2001年,页31-43。Kluwer学术/充气出版商,纽约,纽约,美国。gydF4y2Ba

93年。gydF4y2BaKalafatis, M。Negrescu, E., Byzova, T., and Plow, E.F. Platelets and prothrombin. In:血小板功能:评估、诊断和功能。gydF4y2BaEds。m·奎因和d·菲茨杰拉德;2005年,页279 - 296。胡玛纳媒体公司,风险中,新泽西,美国。gydF4y2Ba